OK, here’s the scenario:

Meal 1:  Two large pieces deep-fried batter-coated chicken (breast and
drumstick); a large serving of batter-fried mushrooms (a little ketchup
as condiment); about one and a half cups of mashed potatoes with gravy;
a biscuit with a liberal amount of butter and honey; a side salad
lightly dressed with Olive Oil and Wine-Garlic vinegar; 17-ounce
regular Coke.

My glucose before eating:  95
1 Hour after finishing meal:  114
2 Hour: 95
3 Hour: 107
4 Hour: 100
5 Hour:  95

Meal 2 (different day):  (Peter Pan Chunky)Peanut Butter and Strawberry
Jam sandwich on thinly-sliced high-fiber whole wheat bread (I didn’t
use large amounts of either of the fillings); bowl of creamy tomato
soup (condensed + water); 6 or 7 large Tostitos tortilla chips;
17-ounce regular Coke.

Before eating:  96
1 Hour after finishing meal:  154
2 Hour:  139
3 Hour:  133
4 Hour:  124
5 Hour:  86

I’ve found that eating meat with a meal seems to really help
normalize my post-meal readings, but considering how big Meal 1 was and
how filled it was with "bad stuff", I’m perplexed that the two
pieces of (batter-fried!) chicken could still have such a
"normalizing" effect that lasted the full five hours.  Any thoughts
(other than chiding me for eating so poorly)?   Was the 86 five hours
after Meal 2 a hypoglycemic reaction of sorts?  Is the sweetness of the
Peanut Butter and Jelly possibly the culprit in Meal 1 (the Coke was
common to both meals)?

I’ve seen the same pattern I listed for Meal 1 if I have, say, a big
steak dinner with loads of sourdough bread & butter, mashed potatoes,
big salad with regular cheese-based dressing, and regular Coke, and
sometimes even a dessert like creme brulee:  My blood sugar just
doesn’t stray very far from fasting level.  As for something like
pasta — it can send my blood sugar soaring as high as 170 if eaten
without meat, but if I have, say, a pork chop with it (e.g., if I’m
at a restaurant sharing entrees with my wife), I can see a bg pattern
similar to Meal 1.  I.e., not just a "big" difference from the
pasta-but-no-meat, but rather a *huge* difference.

I don’t take any db meds.  Thanks in advance for any thoughts.

Lee

Can anyone point me to a web site that explains the abbreviations on blood
work lab results? Thanks.

Families of fat women face slimmer pay – study
25 May 2005 21:52:33 GMT

Source: Reuters

WASHINGTON, May 25 (Reuters) – Heavy-set women are likely to face worse
socioeconomic outcomes than slimmer colleagues, but fat men do just fine, a
new research study has found.

"Body mass significantly decreases women’s family income," the study by two
researchers at New York University found. "However … men experience no
negative effects of body mass on economic outcomes."

Dalton Conley, director of NYU’s Center for Advanced Social Science
Research, and NYU graduate student Rebecca Glauber found that a 1 percent
increase in a woman’s body mass index — a measure of weight relative to
height — pushes family income down by about 0.6 percent.

Similarly, the researchers found a woman’s "occupational prestige," a
measure of the social status of differing jobs, also dropped as body mass
rose, although to a somewhat lesser degree: 0.4 percent for each 1 percent
increase in body mass.

Conley said the marriage market appeared to account for most of the
differences in body mass-related outcomes among women.

"Women who are heavier for their height tend to have lower chances of
getting married in the first place. If they do get married they tend to
marry spouses who have less earning power and they also have a higher
likelihood of getting divorced," he said. "All those three factors reduce
their total family income."

Conley and Glauber also found that the conventional wisdom that tall men
were more successful than shorter men did not hold true. "The talk is that
height for men is what slimness is for woman, but it turns out there is
absolutely no effect," Conley said.

The study, which was recently posted to the Web site of the prestigious
National Bureau of Economic Research, did not definitively rule out the
possibility that a lower socioeconomic position to begin with might lead to
a high body mass reading.

But Conley said the research, which compared outcomes between sisters and
between brothers, suggested this was unlikely.

As for men, body mass appeared to have little impact.

"Body mass does not reduce their economic status outcomes, it does not
reduce their likelihood of marriage, and it does not increase their
likelihood of divorce, separation, or widowhood," the researchers wrote.

Just replied to a message and got a note from Google that I had been
cross-posted to a bunch of other groups.  The original had to do with
fat women being paid less than thinner women.  I thought that was a
silly study since common sense would give the same conclusion.  Now I’m
replying to a bunch of feminist groups (I guess, from the sounds of the
titles!)  Oh well, I can handle that, but I don’t remember seeing any
warning that the original poster was cross-posting.
Nan, Type 2 since 1990 or so

The American Diabetic Association (ADA) is suddenly countering decades
of scientific studies that have consistently linked diets high in sugar
to diabetes. In a May 16 interview, Richard Kahn, the chief scientific
and medical officer with the ADA said "What is the evidence that sugar
itself has anything to do with diabetes? There is no evidence."
Coincidentally, last month, the ADA announced a "three-year,
multi-million dollar alliance" with Cadbury Schweppes, which is the
third largest producer of soft drinks in the world.
http://www.organicconsumers.org/school/diabetes051705.cfm

I’ve just got a mass-letter from LifeScan as I am "registered as a user
of a OneTouch Ultra" (though registration is as far as it goes – the
device itself is lying in a dusty corner somewhere).

They admit that the BS units shown (mmol/L or mg/dL) often get flipped
when people enter the date and time, and they blame the users for this.
They also admit that this sometimes happens "e.g. when the device falls
down during measurement".

The consequence of this is that a perfectly reasonable measurement like
110 (mg/dL) ends up being displayed as 6.1 (mmol/L).  Of course, the
decimal point on the screen is barely noticeable, so the poor bloody
diabetic reads a value of 61 when it’s really 110.  Not good.

And LifeScan’s recommendation?  Check the units every time you use the
thing.  Wow!  Talk about being user friendly.

And, as a publicity stunt, they’re offering to exchange double-unit
meters for fixed mmol/L ones, but not the other way around, for anybody
who uses mmol/L to measure with.  Nobody does in Germnay.

But, how about the novel idea of having a BS meter which just measures BS
and does nothing else?  This doesn’t appear to have occurred to them.
Anybody with a decade or more’s experience of technical things knows that
if you pile inessential functionality onto a device, the chance of things
not working properly increases disproportionately.  (Just look at any
Microsoft software apart from Minesweeper to see that.)

Do people really need or want a calendar and wrist watch bundled in with
their BS meter?  I certainly don’t.

And down at the bottom, they have the cheek to write "We’d be delighted
to support you further in your diabetes ‘self-management’".  This is, in
my experience, being somewhat economical with the truth.

–
Alan Mackenzie (Munich, Germany)
Email: a…@muuc.dee; to decode, wherever there is a repeated letter
(like "aa"), remove half of them (leaving, say, "a").

Smart Capsule for Insulin – Polymeric Capsules
28 May 2005

Moscow chemists have suggested a way to produce insulin in pills
instead of injections. They have developed polymeric capsules that
would protect insulin from destructive effect of digestive juices. The
research has been accomplished with financial support of the Ministry
of Industry, Science and Technology of Russia within the framework of
international scientific cooperation.

Once insulin appeared, diabetes mellitus is no longer a verdict for the
patients. However, to control the sugar level in blood, diabetics have
to make injections on a regular basis up to several times a day, which
undoubtedly makes their life more difficult. The lack of timely
injection may result in fatal outcome. The pills would significantly
simplify their life. However, the fact that insulin gets quickly
destroyed when it appears in acid medium of the stomach does not
exclude but significantly complicates creation of pilled insulin.
Researchers of the Chemical Faculty, Lomonosov Moscow State University,
have found the way to protect insulin from digestive juices’
destructive effect and to preserve the ability to perform its function.

To introduce insulin in the organism, the Russian chemists suggested
that multi-layer polymeric capsules should be used. These polymeric
capsules are stable and remain intact in acid medium, and in neutral
medium, capsules gradually excrete insulin.

To create such capsules, the researchers used two polymers – positive
protamin and negative dextransulphate. They formed layers in series one
upon the other according to the plus towards minus principle and made a
multi-layer covering around the insulin filling, which makes up to 85
percent of the entire microparticle.

Insulin covered by protective capsule is stable at pH from 1.7 to 5
units, when pH increases higher than 5 units, insulin gets released.
Further pH increase up to 8 units results in accelerated protein
release rate. Such behavior of particles occurs due to the fact that at
pH higher than 5.5 insulin acquires negative charge and its bond with
the negatively charged polymer of the first layer – dextransulphate –
gets destroyed.

Such pH-dependence of protective polymeric capsules provides
fundamental capability to create insulin in pills. In the stomach,
where medium is extremely acid, these capsules would protect the
insulin molecule and would not allow its destruction. Having gone
through the stomach and having reached the small intestine and ileum,
where pH reaches 6 to 8 units, capsules will start to excrete insulin
intensely. In thin intestines, insulin can penetrate blood. Therefore,
the capsule determines by itself where insulin should be retained in
closed form and where it should be released.

The polymers used to create capsules belong to natural biodegradable
polymers. After utilization they get easily destroyed by enzymes and
removed from the organism without causing any harm to health.

Reference URL
http://www.informnauka.ru

SOURCE: http://www.alphagalileo.org

I see so much concern over injected insulin.  I have never found
it to be a real problem except the storage and handling
of the junk.

The injection method has a long history and the
experiences with it.   The mechanism is very predictable.

There have many efforts to find a pill as long as I have
had diabetes.  None have ever worked well or did not
work at all.

I just shot a blood sugar and injected.  The only
problem was remembering to do each step.  Use
a check list now.

We need more effort in finding a method to sense
and inject insulin as needed.  For many of us the
complications are very closely tied to high
blood sugars.

This work has never been done by a profit firm
since it will be risky and long term.  No profit
very soon so put the money elsewhere.
A nominal effort, just in case.

So needles will be around for a long time.
Relax and make them your friend—saving your life.

Why has a large reward system not been made for diabetes
achievements.   No result–no money.l
                                     Guy.

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Hi All

I came across this study when I was researching something
else – as usual:-)

The link goes straight to the full PDF file. Hopefully the
conclusions should be enough to interest some to read
further.

Postchallenge Plasma Glucose and Glycemic Spikes Are More
Strongly Associated With Atherosclerosis Than Fasting
Glucose or HbA1c Level
http://care.diabetesjournals.org/cgi/reprint/23/12/1830.pdf

"OBJECTIVE- To observe the relationship of fasting plasma
glucose (FPG), postchallenge plasma glucose (PG) (30, 60,
90, and 120 min during an oral glucose tolerance test
[OGTT], as well as maximal PG during an OGTT, postchallenge
glucose spikes [PGS], and glucose under the OGTT curve), and
HbA1c to intima-media thickness (IMT) as a marker of
atherosclerosis.

RESEARCH DESIGN AND METHODS- OGTT, ultrasound measurement of
carotid IMT, and various atherosclerosis risk factors, such
as family history of diabetes, obesity, and/or
hyperlipoproteinemia, but without known diabetes, were
analyzed in 582 individuals aged 40-70 years and at risk for
type 2 diabetes.

RESULTS- In univariate analysis, all examined glycemic
parameters were significantly correlated to IMT. The 2-h
postchallenge plasma glucose showed the strongest odds ratio
(OR) of 1.88 (1.34-2.63) in relation to abnormal IMT. All PG
variables, except for 30-min glucose in OGTT, showed a
significant OR, whereas the OR for HbA1c and FPG was not
significant. In logistic regression analysis, 2-h PG was
identified as the strongest determinant of IMT from all
glycemic parameters. The 2-h PG and PGS, but not FPG, were
associated with a significant rise of IMT in tertiles of
HbA1c. Glycemic parameters were strongly related to each
other and to many atherosclerosis risk factors. In
multivariate analysis including a variety of atherosclerosis
risk factors, 2-h PG was a significant independent
determinant of IMT.

CONCLUSIONS- PG and PGS are more strongly associated with
carotid IMT than FPG and HbA1c level and modify
substantially the risk for atherosclerosis, estimated by
HbA1c alone, in a cohort at risk for diabetes and in the
early diabetes stage."

Diabetes Care 23:1830-1834, 2000

Cheers Alan, T2, Australia.
—
Everything in Moderation – Except Laughter.

Has anyone heard of or tried Ambrotose?